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nf κb agonist  (MedChemExpress)


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    MedChemExpress nf κb agonist
    Nf κb Agonist, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 95/100, based on 94 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/nf+%CE%BAb+agonist/Phorbol/pm41825725-224-12-28
    Average 95 stars, based on 94 article reviews
    nf κb agonist - by Bioz Stars, 2026-09
    95/100 stars

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    Article Title: Liraglutide promotes osteogenic differentiation of mesenchymal stem cells by inhibiting M1 macrophage polarization and CXCL9 release in vitro.
    Article Snippet: As a GLP-1 receptor agonist widely used in treating type 2 diabetes, liraglutide shows potential applications in bone tissue engineering.. This study investigated liraglutide’s direct effects on rat bone marrow mesenchymal stem cells (BMSCs) osteogenic differentiation and its regulatory mechanism through macrophage polarization.. Results showed that liraglutide significantly enhanced BMSC migration and osteogenic differentiation.

    Article Title: Da-Cheng-Qi Decoction attenuates inflammatory edema and endoplasmic reticulum stress in acute pancreatitis via suppressing NF-κB pathway.
    Article Snippet: 23 Ethnopharmacological relevance: Da-Cheng-Qi Decoction (DCQD) is clinically used for acute pancreatitis (AP).. AP is 24 an inflammatory disorder, characterized by inflammatory edema (IE) and endoplasmic reticulum stress (ERS).. 25 Aim of the study: This study aims to investigate whether DCQD attenuates IE and ERS in AP via nuclear factor κB 26 (NF-κB) pathway.



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    EVs attenuate lipopolysaccharide-induced NF-κB <t>p65</t> activation and nuclear export. ( A ) The protein levels of phosphorylated-p65 and IκBα in AC16 cells treated with LPS and EVs were assessed with Western blot ( n = 3, biological replicates). ( B ) The protein level of p65 in the cytoplasm or nucleus after stimulation with EVs in AC16 cells treated with LPS was assessed with Western blot ( n = 3, biological replicates). *, p < 0.05; **, p < 0.01.
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    EVs attenuate lipopolysaccharide-induced NF-κB p65 activation and nuclear export. ( A ) The protein levels of phosphorylated-p65 and IκBα in AC16 cells treated with LPS and EVs were assessed with Western blot ( n = 3, biological replicates). ( B ) The protein level of p65 in the cytoplasm or nucleus after stimulation with EVs in AC16 cells treated with LPS was assessed with Western blot ( n = 3, biological replicates). *, p < 0.05; **, p < 0.01.

    Journal: Current Issues in Molecular Biology

    Article Title: Plasma-Derived Extracellular Vesicles Inhibit Lipopolysaccharide-Induced Apoptosis and Oxidative Stress in Human AC16 Cardiomyocytes

    doi: 10.3390/cimb48020174

    Figure Lengend Snippet: EVs attenuate lipopolysaccharide-induced NF-κB p65 activation and nuclear export. ( A ) The protein levels of phosphorylated-p65 and IκBα in AC16 cells treated with LPS and EVs were assessed with Western blot ( n = 3, biological replicates). ( B ) The protein level of p65 in the cytoplasm or nucleus after stimulation with EVs in AC16 cells treated with LPS was assessed with Western blot ( n = 3, biological replicates). *, p < 0.05; **, p < 0.01.

    Article Snippet: AC16 cardiomyocytes (EK-Bioscience, Shanghai, China), lipopolysaccharide (LPS, Escherichia coli O111:B4; Sigma-Aldrich, St. Louis, MO, USA), RIPA lysis buffer (KeyGEN, Nanjing, China), protease/phosphatase inhibitor cocktail (KeyGEN, Nanjing, China), BCA Protein Assay Kits (Thermo Scientific, Waltham, MA, USA), NF-κB p65 agonist (Betulinic acid, Selleck, Houston, TX, USA), Nuclear and Cytoplasmic Protein Extraction Kits (Abcam, Cambridge, MA, USA), TUNEL FITC Apoptosis Detection Kits (Vazyme, Nanjing, China), and ROS Assay Kits (Beyotime, Shanghai, China) were used.

    Techniques: Activation Assay, Western Blot

    The NF-κB p65 agonist eliminates the anti-apoptosis effects of EVs. ( A ) The apoptosis of AC16 cells treated with LPS with or without EVs or the NF-κB agonist was evaluated by flow cytometry ( n = 4, biological replicates). ( B ) The protein levels of Bax, Bcl-2 and Caspase3 in AC16 cells treated with LPS with or without EVs or the NF-κB agonist were evaluated by Western blot ( n = 3, biological replicates). ( C ) The ROS level in AC16 cells treated with LPS with or without EVs or the NF-κB agonist was evaluated by flow cytometry ( n = 4, biological replicates). *, p < 0.05; **, p < 0.01.

    Journal: Current Issues in Molecular Biology

    Article Title: Plasma-Derived Extracellular Vesicles Inhibit Lipopolysaccharide-Induced Apoptosis and Oxidative Stress in Human AC16 Cardiomyocytes

    doi: 10.3390/cimb48020174

    Figure Lengend Snippet: The NF-κB p65 agonist eliminates the anti-apoptosis effects of EVs. ( A ) The apoptosis of AC16 cells treated with LPS with or without EVs or the NF-κB agonist was evaluated by flow cytometry ( n = 4, biological replicates). ( B ) The protein levels of Bax, Bcl-2 and Caspase3 in AC16 cells treated with LPS with or without EVs or the NF-κB agonist were evaluated by Western blot ( n = 3, biological replicates). ( C ) The ROS level in AC16 cells treated with LPS with or without EVs or the NF-κB agonist was evaluated by flow cytometry ( n = 4, biological replicates). *, p < 0.05; **, p < 0.01.

    Article Snippet: AC16 cardiomyocytes (EK-Bioscience, Shanghai, China), lipopolysaccharide (LPS, Escherichia coli O111:B4; Sigma-Aldrich, St. Louis, MO, USA), RIPA lysis buffer (KeyGEN, Nanjing, China), protease/phosphatase inhibitor cocktail (KeyGEN, Nanjing, China), BCA Protein Assay Kits (Thermo Scientific, Waltham, MA, USA), NF-κB p65 agonist (Betulinic acid, Selleck, Houston, TX, USA), Nuclear and Cytoplasmic Protein Extraction Kits (Abcam, Cambridge, MA, USA), TUNEL FITC Apoptosis Detection Kits (Vazyme, Nanjing, China), and ROS Assay Kits (Beyotime, Shanghai, China) were used.

    Techniques: Flow Cytometry, Western Blot